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Keywords: Copper, Cuproptosis, Reactive oxygen species, Oxidative stress Introduction As copper (Cu) is a trace element in the human body, its intracellular concentration is maintained at a very low level by evolutionarily conserved homeostatic mechanisms that prevent the accumulation of free copper within the cells

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Results Dexamethasone exposure reduces late ferroptosis To better understand the effect of dexamethasone on ferroptosis in renal pathophysiology, we first leveraged the human-derived renal tubular cell line CD10-135, which has been demonstrated to recapitulate key features of its origin [14]